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At one end of the spectrum, metabolic dysfunction-associated steatotic liver (hepatic steatosis, MASL) may progress to a more advanced state called metabolic dysfunction-associated steatohepatitis (MASH) characterized by significant hepatocyte damage, inflammation, and pericellular fibrosis, and further to cirrhosis (Friedman et al
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DHCA together with dihydroferulic and dihydroxyphenylacetic acids were only 3 out of 18 polyphenol compounds that were able to inhibit at least 50% of the prostaglandin E2 formed by colon fibroblast cells (CCD-18) after their stimulation with interleukin-1β (IL-1b) [98]
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