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In light of the ability of either half of the kinase domain to bind FAK, and our previous data showing the PH domain to confer FAK specificity amongst Akt isoforms, this region of Akt1 presents a promising target for disrupting FAK-specific binding
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643 Signaling pathways influenced by ferroptosis are implicated in numerous pathological conditions, and their role in depression is likely intricate and multifaceted
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