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The N‐terminal p53 transactivation domain, for example, is largely disordered but possesses two binding motifs with α‐helical propensity, named TAD1 (residues 17–29) and TAD2 (residues 40–57)
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IL-9/STAT3/fatty acid oxidation-mediated lipid peroxidation contributes to Tc9 cell longevity and enhanced antitumor activity
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