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Our study demonstrates the efficacy of dual-pathway delivered non-gene-edited hCiPSC-NSCs in alleviating ASD-like phenotypes
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These interacting factors converge on acetaldehyde-adduct formation, redox imbalance, mitochondrial dysfunction, inflammation, altered neurochemical signalling, and impaired cellular repair, thereby producing heterogeneous hepatic, neurological, carcinogenic, developmental, and behavioral outcomes
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